Asbestos : risks, environment and impact / Antonio Soto and Gael Salazar, editors.

Format:

Book

Contributor:

Soto, Antonio, 1961-

Salazar, Gael.

Series:

Environmental Health - Physical, Chemical and Biological Factors

Environmental health--physical, chemical and biological factors series

Language:

English

Subjects (All):

Asbestos--Toxicology.

Asbestos.

Lungs--Dust diseases.

Lungs.

Environmentally induced diseases.

Physical Description:

1 online resource (217 p.)

Edition:

1st ed.

Place of Publication:

New York : Nova Biomedical Books, c2009.

Language Note:

English

Summary:

Asbestos is a naturally occurring silicate mineral with long, thin fibrous crystals. It is known as the miracle mineral because of its soft and pliant properties, as well as its ability to withstand heat. Asbestos is known to be toxic and the inhalation of asbestos can be potentially lethal. Since the mid 1980s, many uses of asbestos have been banned in several countries. The Environmental Protection Agency also banned the use of asbestos and as a result, man-made fibers have been manufactured and used in many applications to replace asbestos. However, asbestos can still be found naturally in the air outdoors and in some drinkable water, including water from natural sources. As this book discusses, studies conducted in laboratory animals have shown that certain man-made fibers may have biological effects similar to those of asbestos. This book addresses this serious health concern caused by asbestos as well as potential fiber-related lung injury due to the inhalation of man-made fiber aerosols.

Contents:

Intro

ASBESTOS: RISKS,ENVIRONMENT AND IMPACT

CONTENTS

PREFACE

DEPOSITION OF MAN-MADE FIBERS IN THE HUMANRESPIRATORY AIRWAY

ABSTRACT

1.1. INTRODUCTION

1.2. EXPERIMENTAL METHOD

1.2.1. Human Nasal Airway Cast

1.2.2. Human Respiratory Airway Casts

1.2.3. Fiber Materials

1.2.4. Experimental Setup

1.2.5. Sample Preparation, Fiber Counting, and Length Measurement

1.3. RESULTS AND DISCUSSION

1.3.1. Fiber Deposition in the Human Nasal Airway

1.3.2. Fiber Deposition Efficiency in the Human Nasal Airway

1.3.3. Comparison of Nasal Deposition between Fibers and Compact Particles

1.3.4. Empirical Model for Fiber Deposition in the Nasal Airway

1.3.5. Fiber Deposition in the Human Oral Airway

1.3.6. Fiber Deposition Pattern in the Human Respiratory Airway

1.3.7. Fiber Deposition in the Tracheobronchial Airways

1.3.8. Comparison of Fiber Deposition in the Human Airways

1.4. CONCLUSION

REFERENCES

NUMBER OF PLASMIDS TRANSPORTED INTOESCHERICHIA COLI THROUGH THE YOSHIDA EFFECTAND PREDICTED STRUCTURE OF THEPENETRATION-INTERMEDIATE

INTRODUCTION

MATERIALS AND METHODS

E. COLI STRAIN AND PLASMIDS

PREPARATION OF THE CHRYSOTILE COLLOIDAL SOLUTION

STANDARD PROTOCOL OF HYDROGEL EXPOSURE

INTRODUCTION OF MULTIPLE PLASMIDS INTO E. COLI CELLS USINGTHE HYDROGEL EXPOSURE METHOD

PLASMID STABILITY

NUMBER OF PLASMIDS INTRODUCED INTO EACH PENETRATIONINTERMEDIATEUSING THE HYDROGEL EXPOSURE METHOD

PREDICTED STRUCTURE OF A PENETRATION-INTERMEDIATE

RESULTS

INTRODUCTION OF MULTIPLE PLASMIDS INTO E. COLI USINGTHE HYDROGELEXPOSURE METHOD

NUMBER OF PLASMIDS INTRODUCED INTO THE PENETRATIONINTERMEDIATEUSING THE HYDROGEL EXPOSURE METHOD

PREDICTED STRUCTURE OF THE PENETRATION-INTERMEDIATE

CONCLUSION

REFERENCES.

PREVENTION AND DETECTION OF ASBESTOSRELATEDDISEASES IN FINLAND

USE OF ASBESTOS AND EXPOSURE TO ASBESTOS

Monitoring the Health Situation of those Who Have Been Exposedto Asbestos

DISEASES CAUSED BY ASBESTOS AND MORTALITY FROM ASBESTOSRELATEDDISEASES

Lung Cancer

Mesothelioma

Asbestosis

Pleural Diseases

THE ASBESTOS EPIDEMIC - IS IT OVER?

Early Lung Cancer Diagnostics

Mesothelioma Panel

Expert Groups in Pneumoconiosis Diseases

Registered Asbestos-Related Diseases

INTERNATIONAL COLLABORATION

BIBLIOGRAPHY

AUTOANTIBODY PROFILES OF AN ASBESTOSEXPOSEDPOPULATION

AMPHIBOLE ASBESTOS CONTAMINATION INLIBBY, MT

Sample and Data Collection

Detection of Serum Autoantibodies

Addressable Laser Bead Immunosorbent Assay (ALBIA)

Quantification of IgM Rheumatoid factor (RF), Anti-Cyclic CitrullinatedPeptide (CCP), Topo I, and Histone by ELISA

Cell-Based ELISA for Anti-Fibroblast Antibodies

Statistical Analysis

Frequency of Antinuclear Antibodies

Autoantibody Characterization

IgM RF and Anti-CCP Frequency

Correlation of Autoantibodies with Extent of Exposure and ARD

Presence of Antibodies to Fibroblasts

DISCUSSION

ACKNOWLEDGEMENTS

EPIDEMIOLOGY OF MESOTHELIOMA:THE ROLE OF ASBESTOS

DEFINITION

DESCRIPTIVE EPIDEMIOLOGY OF MESOTHELIOMA

Incidence

Mortality

Survival

Temporal Trends

Geographic Distribution

ETIOLOGICAL ROLE OF ASBESTOS AND OTHER FIBERS

Asbestos

Other Mineral Fibers Implicated in the Etiopathogenesis of Mesotheliomas

Duration and Intensity of Exposure to Asbestos and Dose-Response Relation

Occupational Exposure

a) Direct Occupational

b) Indirect Occupational

c) Environmental Occupational.

DESCRIPTION OF TYPES OF EXPOSURE AND RISK IN VARIOUSOCCUPATIONAL SECTORS

Production of Asbestos Cement

Insulation and Maintenance Activities of Insulated Industrial Plants

Construction, Maintenance, Restoration and Repair of Ships

Construction, Maintenance, Restoration and Repair of Railway Carriages

Personnel Travelling in Means of Transportation Insulated with Asbestos:Ships and Trains

Industrial and Civilian Construction Work

Textile Sector

Installation and Repair of Lifts

ENVIRONMENTAL EXPOSURE

Urban Environment

CONTRIBUTION OF THE EPIDEMIOLOGY OFMESOTHELIOMA TO PUBLIC HEALTH INITIATIVES

Environmental Reclamation and Identification of Groups at Risk

Prevention of the Asbestos Pathology in Developing Countries

ASBESTOS LITIGATION: PROSPECTSFOR LEGISLATIVE RESOLUTION*

SCOPE OF LITIGATION

PROCEDURAL IMPROVISATION

POLICY ALTERNATIVES

Status Quo

Changes in Tort Law

Medical Criteria

Administrative System

THE TRUST FUND APPROACH

Funding Adequacy

Compensation Adequacy

Diagnostic Categories

Disputed Categories

The Tobacco Question

Diagnostic Quality Control

Transition Issues

Pending Cases

Pending Settlements

Funding Schedule

SUNSET

THE EFFICIENCY OF THE CUSCORE TEST ASCOMPARED TO THAT APPLIED TO SMR INDETECTION OF A CARCINOGENIC EXPOSURE

BACKGROUND

THE TIME INTERVAL BETWEEN EVENTS

THE CUSCORE TEST

THE EFFICIENCY OF THE CUSCORE TEST IN ACONCRETE EXAMPLE

IMPROVING THE TEST EFFICIENCY

RETROPERITONEAL FIBROSIS - A NEW ASBESTOSRELATEDDISEASE

HISTORY AND EPIDEMIOLOGY

CLINICAL PICTURE AND TREATMENT

AETIOLOGY

RETROPERITONEAL FIBROSIS AND ASBESTOS EXPOSURE

RETROPERITONEAL AND PLEURAL FIBROSIS

CONCLUSIONS.

ENDEMIC PLEURAL PLAQUES AND MESOTHELIOMASIN NORTHERN GREECE

METHODS AND RESULTS

1st Study Phase (1988-1990)

X-ray Study

Environmental Study

A. Petrologic Study

B. Air Contamination Study

2nd Study Phase (2003)

IMMUNOLOGICAL EFFECTS OF ASBESTOS

TEMPORARY HIGH-DOSE EXPOSURE19

CONTINUOUS LOW-DOSE EXPOSURE20

CHARACTERISTICS OF CONT.EXP. MT-2 CELLS

CLINICAL TRANSLATION

FUTURE INVESTIGATIONS

ACKNOWLEDGEMENT

INDEX.

Notes:

Description based upon print version of record.

Includes bibliographical references and index.

ISBN:

1-61209-853-3

OCLC:

923660255