Glutamate : functions, regulation, and disorders / Golda Chayat and Avital Yedidya, editors.

Format:

Book

Contributor:

Chayat, Golda.

Yedidya, Avital.

Series:

Neuroscience Research Progress

Neuroscience research progress

Language:

English

Subjects (All):

Glutamic acid--Metabolism.

Glutamic acid.

Glutamic acid--Physiological effect.

Physical Description:

1 online resource (212 p.)

Edition:

1st ed.

Place of Publication:

New York : Nova Science Publishers, c2012.

Language Note:

English

Summary:

Glutamate is purported to be the most important transmitter for normal brain function. Nearly all excitatory neurons in the central nervous system are glutamatergic, and it is estimated that over half of all brain synapses release this agent. In this book, the authors present current research in the study of the functions, regulation and disorders related to glutamate. Topics include glutamate-scavenging in the treatment of acute brain insults; the physiology of glutamate receptors in schizophrenia; how glutamate works as a neurotransmitter and the role of supraspinal GABA and glutamate in the mediation of pain.

Contents:

""CONTENTS""; ""PREFACE""; ""BLOOD GLUTAMATE SCAVENGING: INSIGHT INTO NEUROPROTECTION""; ""GLUTAMATE AND SCHIZOPHRENIA""; ""EXTRACELLULAR OSMOLARITY MODULATES GLUTAMATE UPTAKE AND RELEASE IN NEURONS, ASTROCYTES AND SYNAPTOSOMES""; ""GLUTAMATE:AGLOBAL REGULATOR""; ""CALCIUM-CALMODULIN KINASE TYPE II-MEDIATED GLUTAMATERGIC PLASTICITY UNDERLIES EXPRESSION OF BENZODIAZEPINE-WITHDRAWAL ANXIETY""; ""THE ROLE OF SUPRASPINAL GABA AND GLUTAMATE IN THE MEDIATION ANDMODULATION OF PAIN""; ""GLUTAMATE RECEPTORS OF THE KAINATE TYPE:AN ION CHANNEL BECOMING AMETABOTROPIC RECEPTOR""; ""INDEX""

Notes:

Description based upon print version of record.

Includes bibliographical references and index.

Description based on print version record.

ISBN:

1-61942-567-X

OCLC:

923653546