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Colloquium on neurobiology of pain / [edited by A. James Hudspeth and Masakazu Konishi].

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Format:
Book
Conference/Event
Author/Creator:
Proceedings of the National Academy of Sciences, Corporate Author.
Conference Name:
Colloquium on Neurobiology of Pain (1999 : Irvine, Calif)
Series:
National Academy of Sciences colloquium series.
National Academy of Sciences colloquium series
Language:
English
Subjects (All):
Neurosciences--Congresses.
Neurosciences.
Pain--Physiological aspects--Congresses.
Pain.
Physical Description:
1 online resource (144 p.)
Edition:
1st ed.
Place of Publication:
Washington, D.C. : National Academy of Sciences, 1999.
Language Note:
English
Contents:
COLLOQUIUM ON NEUROBIOLOGY OF PAIN
NATIONAL ACADEMY OF SCIENCES Colloquium Series
List of Attendees
Contents
The neurobiology of pain
John C. Liebeskind (1935-1997): A tribute
Sodium channels and pain
HYPEREXCITABILITY IN DRG CELLS AFTER INJURY
MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS
SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS
PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY
NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS
SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELS
SODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH
A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of...
EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY
THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY?
CONCLUSIONS
Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia
WHY FOCUS ON NA+ CHANNELS?
WHY TTX-RESISTANT CHANNELS?
Calcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons
RESULTS
DISCUSSION
Ion channels gated by heat
Causalgia, pathological pain, and adrenergic receptors
Forebrain mechanisms of nociception and pain: Analysis through imaging
A visceral pain pathway in the dorsal column of the spinal cord
ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION
MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM
BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY
FUNCTIONAL MRI STUDIES IN MONKEYS
The spinal biology in humans and animals of pain states generated by persistent small afferent input
BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY.
ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE
CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES
Spinal Pharmacology of Facilitated Processing
SYSTEM INTERACTIONS
HUMAN SPINAL PROCESSING
Supraspinal contributions to hyperalgesia
Neurotrophins and hyperalgesia
Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptors
Pain perception: Is there a role for primary somatosensory cortex?
Implications of immune-to-brain communication for sickness and pain
IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS
PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION
Role of Spinal Cord Glia in Exaggerated Pain
CONCLUSIONS AND IMPLICATIONS
Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord
The postnatal development of spinal sensory processing
Transcriptional and posttranslational plasticity and the generation of inflammatory pain
STATE-DEPENDENT SENSORY PROCESSING IN THE SOMATOSENSORY SYSTEM
CONCLUSION
Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions
Does a neuroimmune interaction contribute to the genesis of painful peripheral neuropathies?
Distinct neurochemical features of acute and persistent pain
PREPROTACHYKININ-A PRODUCTS AND ACUTE PAIN
PROTEIN KINASE C Γ AND PERSISTENT PAIN
The genetic mediation of individual differences in sensitivity to pain and its inhibition
THE SCOPE OF INDIVIDUAL VARIABILITY TO PAIN AND ANALGESIA
HERITABILITY OF PAIN-RELATED TRAITS
GENETIC CORRELATIONS AMONG PAIN PHENOTYPES
IDENTIFICATION OF PAIN-RELATED GENES
SEX-SPECIFIC GENETIC MEDIATION OF PAIN AND ANALGESIA
ALLELIC VARIANTS OF PAIN-RELATED GENES.
FUTURE DIRECTIONS
The u opiate receptor as a candidate gene for pain: Polymorphisms, variations in expression, nociception, and opiate…
NAS COLLOQUIA
SCHEDULED
COMPLETED
1999 ORDER FORM.
Notes:
Bibliographic Level Mode of Issuance: Monograph
Includes bibliographical references.
ISBN:
1-280-21036-2
9786610210367
0-309-56933-8
OCLC:
832215282

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