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Colloquium on neurobiology of pain / [edited by A. James Hudspeth and Masakazu Konishi].
- Format:
- Book
- Conference/Event
- Author/Creator:
- Proceedings of the National Academy of Sciences, Corporate Author.
- Conference Name:
- Colloquium on Neurobiology of Pain (1999 : Irvine, Calif)
- Series:
- National Academy of Sciences colloquium series.
- National Academy of Sciences colloquium series
- Language:
- English
- Subjects (All):
- Neurosciences--Congresses.
- Neurosciences.
- Pain--Physiological aspects--Congresses.
- Pain.
- Physical Description:
- 1 online resource (144 p.)
- Edition:
- 1st ed.
- Place of Publication:
- Washington, D.C. : National Academy of Sciences, 1999.
- Language Note:
- English
- Contents:
- COLLOQUIUM ON NEUROBIOLOGY OF PAIN
- NATIONAL ACADEMY OF SCIENCES Colloquium Series
- List of Attendees
- Contents
- The neurobiology of pain
- John C. Liebeskind (1935-1997): A tribute
- Sodium channels and pain
- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY
- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS
- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS
- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY
- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS
- SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELS
- SODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH
- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of...
- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY
- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY?
- CONCLUSIONS
- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia
- WHY FOCUS ON NA+ CHANNELS?
- WHY TTX-RESISTANT CHANNELS?
- Calcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons
- RESULTS
- DISCUSSION
- Ion channels gated by heat
- Causalgia, pathological pain, and adrenergic receptors
- Forebrain mechanisms of nociception and pain: Analysis through imaging
- A visceral pain pathway in the dorsal column of the spinal cord
- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION
- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM
- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY
- FUNCTIONAL MRI STUDIES IN MONKEYS
- The spinal biology in humans and animals of pain states generated by persistent small afferent input
- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY.
- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE
- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES
- Spinal Pharmacology of Facilitated Processing
- SYSTEM INTERACTIONS
- HUMAN SPINAL PROCESSING
- Supraspinal contributions to hyperalgesia
- Neurotrophins and hyperalgesia
- Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptors
- Pain perception: Is there a role for primary somatosensory cortex?
- Implications of immune-to-brain communication for sickness and pain
- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS
- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION
- Role of Spinal Cord Glia in Exaggerated Pain
- CONCLUSIONS AND IMPLICATIONS
- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord
- The postnatal development of spinal sensory processing
- Transcriptional and posttranslational plasticity and the generation of inflammatory pain
- STATE-DEPENDENT SENSORY PROCESSING IN THE SOMATOSENSORY SYSTEM
- CONCLUSION
- Cellular mechanisms of neuropathic pain, morphine tolerance, and their interactions
- Does a neuroimmune interaction contribute to the genesis of painful peripheral neuropathies?
- Distinct neurochemical features of acute and persistent pain
- PREPROTACHYKININ-A PRODUCTS AND ACUTE PAIN
- PROTEIN KINASE C Γ AND PERSISTENT PAIN
- The genetic mediation of individual differences in sensitivity to pain and its inhibition
- THE SCOPE OF INDIVIDUAL VARIABILITY TO PAIN AND ANALGESIA
- HERITABILITY OF PAIN-RELATED TRAITS
- GENETIC CORRELATIONS AMONG PAIN PHENOTYPES
- IDENTIFICATION OF PAIN-RELATED GENES
- SEX-SPECIFIC GENETIC MEDIATION OF PAIN AND ANALGESIA
- ALLELIC VARIANTS OF PAIN-RELATED GENES.
- FUTURE DIRECTIONS
- The u opiate receptor as a candidate gene for pain: Polymorphisms, variations in expression, nociception, and opiate…
- NAS COLLOQUIA
- SCHEDULED
- COMPLETED
- 1999 ORDER FORM.
- Notes:
- Bibliographic Level Mode of Issuance: Monograph
- Includes bibliographical references.
- ISBN:
- 1-280-21036-2
- 9786610210367
- 0-309-56933-8
- OCLC:
- 832215282
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